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Vitamin D supplementation increases osteocalcin but not other markers of bone turnover during short‐term caloric restriction in women
Abstract   Peer reviewed

Vitamin D supplementation increases osteocalcin but not other markers of bone turnover during short‐term caloric restriction in women

DEEPTHA Sukumar, STEPHEN Schneider, HASINA AMBIA‐SOBHAN, YVETTE Schlussel and SUE Shapses
The FASEB journal, v 25(S1), pp 218.5-218.5
Apr 2011

Abstract

Osteocalcin (OC) is a bone matrix gla protein and a marker of bone formation and may be influenced by vitamin D (vit D). Recent reports suggest that OC may also influence glucose homeostasis and act as a regulator of energy metabolism. Vitamin D (vit D) supplementation suppresses parathyroid hormone (PTH) levels, which may attenuate the increased bone turnover (BT) associated with caloric restriction (CR). In addition, vit D may also improve insulin sensitivity in glucose intolerant individuals. In this randomized double blind trial, we examined whether vit D supplementation has differential effects on OC compared to other BT markers in overweight/obese postmenopausal women without evidence of type 2 diabetes, during 6 weeks of CR. Women were supplemented with 2500 IU of vit D3 or placebo, and all received 1.2g of Ca. 42 women (58 ± 6 yrs, 31±4 kg/m2) completed the study and lost 4 ± 1% of weight with no differences between groups. Serum 25‐hydroxyvitamin D (25OHD) increased (7.5 ± 5.8ng/ml) in vit D group (p < 0.01). PTH, propeptide of type 1 collagen, N‐telopeptide of type 1 collagen and pyridinium cross links did not differ, whereas OC was higher in the vit D group (p<0.01). These results show that vit D supplementation during CR influences OC and not other BT markers. To what extent changes in OC are associated with improved insulin sensitivity as opposed to being a marker of bone turnover is currently being examined. Grant Funding Source: NIH‐NIA AG12161

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