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Cancer Metabolism: Cross Talk Between Signaling and O-GlcNAcylation
Book chapter

Cancer Metabolism: Cross Talk Between Signaling and O-GlcNAcylation

Christina M Ferrer and Mauricio J Reginato
Cancer Genomics and Proteomics
20 Jun 2014
PMID: 25030920

Abstract

O-GlcNAc transferase Lactate Hexosamine biosynthetic pathway HIF-1α Metabolism LKB1 Signaling Glucose uptake mTOR O-GlcNAcylation AMPK Hypoxia ATP Cancer
Cancer cells exhibit a unique metabolic shift to aerobic glycolysis that has been exploited diagnostically and therapeutically in the clinic. Oncogenes and tumor suppressors alter signaling pathways that lead to alterations of glycolytic flux. Stemming from glycolysis, the hexosamine biosynthetic pathway leads to elevated posttranslational addition of O-linked-β-N-acetylglucosamine (O-GlcNAc) on a diverse population of nuclear and cytosolic proteins, many of which regulate signaling pathways. This unit outlines techniques used to detect metabolic alterations in cancer cells, regulation by signaling pathways, and cellular O-GlcNAcylation.

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#3 Good Health and Well-Being

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Web of Science research areas
Biochemical Research Methods
Biochemistry & Molecular Biology
Genetics & Heredity
Pathology
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