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A selective human beta(3) adrenergic receptor agonist increases metabolic rate in rhesus monkeys
Journal article   Open access   Peer reviewed

A selective human beta(3) adrenergic receptor agonist increases metabolic rate in rhesus monkeys

M H Fisher, A M Amend, T J Bach, J M Barker, E J Brady, M R Candelore, D Carroll, M A Cascieri, SHL Chiu, L P Deng, …
The Journal of clinical investigation, v 101(11), pp 2387-2393
01 Jun 1998
PMID: 9616210
url
http://www.jci.org/articles/view/2496/files/pdfView
Published, Version of Record (VoR) Open
url
https://doi.org/10.1172/JCI2496View
Published, Version of Record (VoR) Open

Abstract

Life Sciences & Biomedicine Medicine, Research & Experimental Research & Experimental Medicine Science & Technology
Activation of beta(3) adrenergic receptors on the surface of adipocytes leads to increases in intracellular cAMP and stimulation of lipolysis. In brown adipose tissue, this serves to upregulate and activate the mitochondrial uncoupling protein 1, which mediates a proton conductance pathway that uncouples oxidative phosphorylation, leading to a net increase in energy expenditure. While chronic treatment with beta(3) agonists in nonprimate species leads to uncoupling protein 1 up-regulation and weight loss, the relevance of this mechanism to energy metabolism in primates, which have much lower levels of brown adipose tissue, has been questioned. With the discovery of L-755,507, a potent and selective partial agonist for both human and rhesus beta(3) receptors, we now demonstrate that acute exposure of rhesus monkeys to a beta(3) agonist elicits lipolysis and metabolic rate elevation, and that chronic exposure increases uncoupling protein 1 expression in rhesus brown adipose tissue. These data suggest a role for beta(3) agonists in the treatment of human obesity.

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