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Activation of cPLA(2) in vascular smooth muscle
Journal article   Peer reviewed

Activation of cPLA(2) in vascular smooth muscle

Edward F LaBelle and Erzsebet Polyak
Eicosanoids and Other Bioactive Lipids in Cancer, Inflammation, and Radiation Injury, v 4, pp 177-182
01 Jan 1999
PMID: 10667328

Abstract

Biochemistry & Molecular Biology Life Sciences & Biomedicine Medicine, Research & Experimental Research & Experimental Medicine Science & Technology Cell Biology
Force regulation in vascular smooth muscle is under the control of a variety of lipid-dependent second messengers, such as inositol 1,4,5 trisphosphate and diacylglycerol (Hashimoto et al.,1986; Gu et al., 1991). Our laboratory has demonstrated that norepinephrine can induce force in rat tail artery via the stimulation of IP3 production and the resulting release of calcium ions from intracellular stores (Gu et al.,1991; Somlyo et al,1985). It appears that IP3 production in this tissue is under the control of a G-protein-dependent isoform of phospholipase C known as phospholipase Cb2 (LaBelle and Murray, 1990; LaBelle and Polyak,1996). We have also demonstrated that norepinephrine can activate phospholipase D in rat tail artery, with the resulting production of choline and phosphatidic acid from the lipid phosphatidylcholine (Gu et al, 1992).

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Collaboration types
Domestic collaboration
Web of Science research areas
Biochemistry & Molecular Biology
Cell Biology
Medicine, Research & Experimental
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