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Journal article
Acute drivers of neuroinflammation in traumatic brain injury
Neural regeneration research, v 14(9), pp 1481-1489
01 Sep 2019
PMID: 31089036
Featured in Collection : UN Sustainable Development Goals @ Drexel
Abstract
Neuroinflammation is initiated as a result of traumatic brain injury and can exacerbate evolving tissue pathology. Immune cells respond to acute signals from damaged cells, initiate neuroinflammation, and drive the pathological consequences over time. Importantly, the mechanism(s) of injury, the location of the immune cells within the brain, and the animal species all contribute to immune cell behavior following traumatic brain injury. Understanding the signals that initiate neuroinflammation and the context in which they appear may be critical for understanding immune cell contributions to pathology and regeneration. Within this paper, we review a number of factors that could affect immune cell behavior acutely following traumatic brain injury.
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Details
- Title
- Acute drivers of neuroinflammation in traumatic brain injury
- Creators
- Kathryn L. Wofford - Drexel UniversityDavid J. Loane - University of Maryland, BaltimoreD. Kacy Cullen - University of Pennsylvania
- Publication Details
- Neural regeneration research, v 14(9), pp 1481-1489
- Publisher
- Wolters Kluwer - Medknow
- Resource Type
- Journal article
- Language
- English
- Academic Unit
- School of Biomedical Engineering, Science, and Health Systems
- Web of Science ID
- WOS:000468060200002
- Scopus ID
- 2-s2.0-85065827019
- Other Identifier
- 991019168117404721
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- Collaboration types
- Domestic collaboration
- International collaboration
- Web of Science research areas
- Cell Biology
- Neurosciences