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Adenosine 5'-triphosphate's role in bradycardia and syncope associated with pulmonary embolism
Journal article   Open access   Peer reviewed

Adenosine 5'-triphosphate's role in bradycardia and syncope associated with pulmonary embolism

Amir Pelleg, Edward S Schulman and Peter J Barnes
Respiratory research, v 19(1), pp 142-142
28 Jul 2018
PMID: 30055609
url
https://doi.org/10.1186/s12931-018-0848-2View
Published, Version of Record (VoR) Open

Abstract

Syncope - diagnosis Syncope - epidemiology Bradycardia - diagnosis Humans Platelet Activation - physiology Pulmonary Embolism - epidemiology Syncope - metabolism Bradycardia - epidemiology Pulmonary Embolism - diagnosis Animals Bradycardia - metabolism Adenosine Triphosphate - physiology Pulmonary Embolism - metabolism
Adenosine 5'-triphiosphate (ATP) is released from cells under physiologic and pathophysiologic conditions. Extracellular ATP acts as an autocrine and paracrine agent affecting various cell types by activating cell surface P2 receptors (P2R), which include trans-cell membrane cationic channels, P2XR, and G protein coupled receptors, P2YR. We have previously shown that ATP stimulates vagal afferent nerve terminals in the lungs by activating P2X2/3R. This action could lead to bronchoconstriction, cough and the local release of pro-inflammatory neuropeptides. In addition, ATP markedly enhances the IgE-dependent histamine release from human lung mast cells. Thus, we have proposed for the first time that extracellular ATP plays a mechanistic role in pulmonary pathophysiology in general and chronic obstructive pulmonary disease (COPD), and acute bronchoconstriction in asthma in particular. The present review examines whether ATP could also play a role in bradycardia and syncope in a subset of patients with pulmonary embolism.

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Web of Science research areas
Respiratory System
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