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Aβ1-28 Fragment of the Amyloid Peptide Predominantly Adopts a Polyproline II Conformation in an Acidic Solution
Journal article   Peer reviewed

Aβ1-28 Fragment of the Amyloid Peptide Predominantly Adopts a Polyproline II Conformation in an Acidic Solution

Fatma Eker, Kai Griebenow and Reinhard Schweitzer-Stenner
Biochemistry (Easton), v 43(22), pp 6893-6898
08 Jun 2004
PMID: 15170326

Abstract

To structurally characterize the nonaggregated state of the amyloid β peptide, which assembles into the hallmark fibrils of Alzheimer disease, we investigated the conformation of the N-terminal extracellular peptide fragment Aβ1-28 in D2O at acidic pD by utilizing combined FTIR and isotropic and anisotropic Raman spectra measured between 1550 and 1750 cm-1. Peptide aggregation is avoided under the conditions chosen. The amide I‘ band was found to exhibit a significant noncoincidence effect in that the first moment of the anisotropic Raman and of the IR band profile appears red-shifted from that of the isotropic Raman scattering. A simulation based on a coupled oscillator model involving all 27 amide I‘ modes of the peptide reveals that the peptide adopts a predominantly polyproline II conformation. Our results are inconsistent with the notion that the monomeric form of Aβ1-28 is a totally disordered, random-coil structure. Generally, they underscore the notion that polyproline II is a characteristic motif of the unfolded state of proteins and peptides.

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Collaboration types
Domestic collaboration
Web of Science research areas
Biochemistry & Molecular Biology
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