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Ca2+ activates human homologous recombination protein Rad51 by modulating its ATPase activity
Journal article   Open access

Ca2+ activates human homologous recombination protein Rad51 by modulating its ATPase activity

Dmitry V Bugreev and Alexander V Mazin
Proceedings of the National Academy of Sciences - PNAS, v 101(27), pp 9988-9993
06 Jul 2004
PMID: 15226506
url
https://doi.org/10.1073/pnas.0402105101View
Published, Version of Record (VoR) Open

Abstract

DNA-Binding Proteins - metabolism Calcium - pharmacology Rad51 Recombinase Sodium Chloride - pharmacology Adenosine Triphosphate - metabolism Adenosine Triphosphatases - metabolism Adenosine Diphosphate - metabolism DNA - metabolism Magnesium - pharmacology
Human Rad51 (hRad51) protein plays a key role in homologous recombination and DNA repair. hRad51 protein forms a helical filament on single-stranded DNA (ssDNA), which performs the basic steps of homologous recombination: a search for homologous double-stranded DNA (dsDNA) and DNA strand exchange. hRad51 protein possesses DNA-dependent ATPase activity; however, the role of this activity has not been understood. Our current results show that Ca(2+) greatly stimulates DNA strand exchange activity of hRad51 protein. We found that Ca(2+) exerts its stimulatory effect by modulating the ATPase activity of hRad51 protein. Our data demonstrate that, in the presence of Mg(2+), the hRad51-ATP-ssDNA filament is quickly converted to an inactive hRad51-ADP-ssDNA form, due to relatively rapid ATP hydrolysis and slow dissociation of ADP. Ca(2+) maintains the active hRad51-ATP-ssDNA filament by reducing the ATP hydrolysis rate. These findings demonstrate a crucial role of the ATPase activity in regulation of DNA strand exchange activity of hRad51 protein. This mechanism of Rad51 protein regulation by modulating its ATPase activity is evolutionarily recent; we found no such mechanism for yeast Rad51 (yRad51) protein.

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Biochemistry & Molecular Biology
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