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Calcium release-activated calcium channels and pain
Journal article   Open access   Peer reviewed

Calcium release-activated calcium channels and pain

Yixiao Mei, James E Barrett and Huijuan Hu
Cell calcium (Edinburgh), v 74, pp 180-185
Sep 2018
PMID: 30096536
url
https://doi.org/10.1016/j.ceca.2018.07.009View
Accepted (AM)Maybe Open Access (Publisher Bronze) Open

Abstract

Calcium release-activated calcium (CRAC) channels are unique among ion channels that are activated in response to depletion of intracellular calcium stores and are highly permeable to Ca compared to other cations. CRAC channels mediate an important calcium signal for a wide variety of cell types and are well studied in the immune system. They have been implicated in a number of disorders such as immunodeficiency, musculosketal disorders and cancer. There is growing evidence showing that CRAC channels are expressed in the nervous system and are involved in pathological conditions including pain. This review summarizes the expression, distribution, and function of the CRAC channel family in the dorsal root ganglion, spinal cord and some brain regions, and discusses their functional significance in neurons and glial cells and involvement in nociception and chronic pain. Although further studies are needed to understand how these channels are activated under physiological conditions, the recent findings indicate that the CRAC channel Orai1 is an important player in pain modulation and could represent a new target for pathological pain.

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33 citations in Scopus

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Collaboration types
Domestic collaboration
Web of Science research areas
Cell Biology
Physiology
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