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Calcium signaling of in situ chondrocytes in articular cartilage under compressive loading: Roles of calcium sources and cell membrane ion channels
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Calcium signaling of in situ chondrocytes in articular cartilage under compressive loading: Roles of calcium sources and cell membrane ion channels

Mengxi Lv, Yilu Zhou, Xingyu Chen, Lin Han, Liyun Wang and X Lucas Lu
Journal of orthopaedic research, v 36(2), pp 730-738
Feb 2018
PMID: 28980722
url
https://doi.org/10.1002/jor.23768View
Published, Version of Record (VoR)Maybe Open Access (Publisher Bronze) Open

Abstract

Animals Calcium Signaling Cartilage, Articular - cytology Cartilage, Articular - metabolism Cattle Cell Membrane - metabolism Chondrocytes - metabolism Endoplasmic Reticulum - metabolism Female Ion Channels - metabolism Male Weight-Bearing
Mechanical loading on articular cartilage can induce many physical and chemical stimuli on chondrocytes residing in the extracellular matrix (ECM). Intracellular calcium ([Ca ] ) signaling is among the earliest responses of chondrocytes to physical stimuli, but the [Ca ] signaling of in situ chondrocytes in loaded cartilage is not fully understood due to the technical challenges in [Ca ] imaging of chondrocytes in a deforming ECM. This study developed a novel bi-directional microscopy loading device that enables the record of transient [Ca ] responses of in situ chondrocytes in loaded cartilage. It was found that compressive loading significantly promoted [Ca ] signaling in chondrocytes with faster [Ca ] oscillations in comparison to the non-loaded cartilage. Seven [Ca ] signaling pathways were further investigated by treating the cartilage with antagonists prior to and/or during the loading. Removal of extracellular Ca ions completely abolished the [Ca ] responses of in situ chondrocytes, suggesting the indispensable role of extracellular Ca sources in initiating the [Ca ] signaling in chondrocytes. Depletion of intracellular Ca stores, inhibition of PLC-IP pathway, and block of purinergic receptors on plasma membrane led to significant reduction in the responsive rate of cells. Three types of ion channels that are regulated by different physical signals, TRPV4 (osmotic and mechanical stress), T-type VGCCs (electrical potential), and mechanical sensitive ion channels (mechanical loading) all demonstrated critical roles in controlling the [Ca ] responses of in situ chondrocyte in the loaded cartilage. This study provided new knowledge about the [Ca ] signaling and mechanobiology of chondrocytes in its natural residing environment. © 2017 Orthopaedic Research Society. Published by Wiley Periodicals, Inc. J Orthop Res 36:730-738, 2018.

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Collaboration types
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Web of Science research areas
Orthopedics
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