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Candida albicans and Candida glabrata clinical isolates exhibiting reduced echinocandin susceptibility
Journal article   Open access   Peer reviewed

Candida albicans and Candida glabrata clinical isolates exhibiting reduced echinocandin susceptibility

Santosh Katiyar, Michael Pfaller and Thomas Edlind
Antimicrobial agents and chemotherapy, v 50(8), pp 2892-2894
Aug 2006
PMID: 16870797
url
https://doi.org/10.1128/AAC.00349-06View
Published, Version of Record (VoR) Open

Abstract

Humans Molecular Sequence Data Peptides, Cyclic - pharmacology Fungal Proteins - antagonists & inhibitors Candidiasis - microbiology Candida albicans - genetics Glucosyltransferases - metabolism Candida albicans - drug effects Candida albicans - isolation & purification Conserved Sequence Genes, Fungal Glucosyltransferases - genetics Antifungal Agents - pharmacology Glucosyltransferases - antagonists & inhibitors Amino Acid Sequence Drug Resistance, Fungal - genetics Fungal Proteins - genetics Sequence Homology, Amino Acid Candida glabrata - isolation & purification Candida glabrata - genetics Candida glabrata - enzymology Microbial Sensitivity Tests - methods Amino Acid Substitution Candida albicans - enzymology Candida glabrata - drug effects Fungal Proteins - metabolism
A recognized hotspot for mutations conferring reduced echinocandin susceptibility (RES) is residue S645 of Candida albicans Gsc1(Fks1). We report that the mutation F641Y is associated with RES in a C. albicans isolate. The analogous Fks2 residue is mutated F to V in a Candida glabrata RES isolate; the introduction of this mutation into susceptible C. glabrata confirmed its role in RES. Y641-equivalent Fks residues were identified in intrinsically RES Fusarium species and Candida guilliermondii.

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Collaboration types
Domestic collaboration
Web of Science research areas
Microbiology
Pharmacology & Pharmacy
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