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Cholecystokinin A and B receptors are differentially expressed in normal pancreas and pancreatic adenocarcinoma
Journal article   Open access   Peer reviewed

Cholecystokinin A and B receptors are differentially expressed in normal pancreas and pancreatic adenocarcinoma

D S Weinberg, B Ruggeri, M T Barber, S Biswas, S Miknyocki and S A Waldman
The Journal of clinical investigation, v 100(3), pp 597-603
01 Aug 1997
PMID: 9239407
url
https://doi.org/10.1172/jci119570View
Published, Version of Record (VoR)Maybe Open Access (Publisher Bronze) Open
url
https://doi.org/10.1172/JCI119570View
Published, Version of Record (VoR) Open

Abstract

Adenocarcinoma - metabolism Biomarkers, Tumor Cholecystokinin - metabolism Humans Pancreas - metabolism Pancreatic Neoplasms - metabolism Receptor, Cholecystokinin A Receptor, Cholecystokinin B Receptors, Cholecystokinin - biosynthesis
Cholecystokinin (CCK) plays an important role in pancreatic carcinogenesis. While human CCK-A and -B receptors have been fully characterized, their relative roles in human pancreatic adenocarcinoma remain unclear. Thus, expression of CCK-A and -B receptors in normal human pancreas, pancreatic adenocarcinomas, and other human extrapancreatic tissues and malignancies was examined, using reverse transcription followed by the polymerase chain reaction (RT-PCR). mRNA isolated from 15 normal pancreas specimens, 22 pancreatic adenocarcinomas, and 58 extrapancreatic tissues and tumors was subjected to RT-PCR using primers specific for human CCK-A and -B receptors. Expression of CCK-B receptors was detected in all tissues arising from pancreas and in most extrapancreatic tissues and tumors. In contrast, CCK-A receptors exhibited a more selective pattern of expression in gall bladder, intestine, brain, ovary, spleen, and thymus. Of significance, CCK-A receptors were expressed selectively in all pancreatic adenocarcinomas, but not in any normal pancreas specimens. In situ hybridization, using receptor-specific riboprobes, localized CCK-A receptor expression to ductal cells, the presumed origin of most human pancreatic adenocarcinomas. Southern blot analysis revealed no evidence of CCK-A receptor gene amplification or rearrangement in pancreatic adenocarcinomas. Because of its selective expression, the CCK-A receptor may serve as selective biomarker for pancreatic adenocarcinoma.

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Collaboration types
Domestic collaboration
Web of Science research areas
Medicine, Research & Experimental
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