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Complete ablation of the 14-3-3epsilon protein results in multiple defects in neuropsychiatric behaviors
Journal article   Open access   Peer reviewed

Complete ablation of the 14-3-3epsilon protein results in multiple defects in neuropsychiatric behaviors

Tomoka Wachi, Brett Cornell and Kazuhito Toyo-oka
Behavioural brain research, v 319, pp 31-36
15 Feb 2017
PMID: 27845227
url
https://europepmc.org/articles/pmc5183496View
Accepted (AM) Open

Abstract

14-3-3 Locomotor activity Memory Neurobehavior Sociability
•The Ywhae homozygous knockout mice had a lower body weight than the wild-type mice.•The complete 14-3-3epsilon deficiency caused increased locomotor activity.•Ywhae homozygous deficient mice showed deficits in cognition.•14-3-3epsilon ablation resulted in increased sociability. Previous studies show that mice with Ywhae deficiency show abnormalities in brain development including defects in neuronal migration of post-mitotic pyramidal neurons as well as neuronal differentiation and proliferation in neuronal progenitor cells. Also, our previous research indicated that the Ywhae knockout mice show moderate defects in working memory and anxiety-like behavior. This previous work was performed using heterozygous mutant mice. Here we performed behavioral analyses using homozygous Ywhae knockout mice and found that the homozygous Ywhae knockout mice have increased locomotor activity, decreased working memory, and increased sociability. Taken together with the results obtained from the previous pathophysiological analyses in the Ywhae knockout mice, the Ywhae knockout mouse is useful for pathophysiological analyses of neuropsychiatric disorders caused by defects during neurodevelopment.

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Collaboration types
International collaboration
Web of Science research areas
Behavioral Sciences
Neurosciences
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