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Current status of inverse agonism at serotonin(2A) (5-HT2A) and 5-HT2C receptors
Journal article   Peer reviewed

Current status of inverse agonism at serotonin(2A) (5-HT2A) and 5-HT2C receptors

V. J. Aloyo, K. A. Berg, U. Spampinato, W. P. Clarke and J. A. Harvey
Pharmacology & therapeutics (Oxford), v 121(2)
01 Feb 2009
PMID: 19109993

Abstract

Life Sciences & Biomedicine Pharmacology & Pharmacy Science & Technology
Contemporary receptor theory was developed to account for the existence of constitutive activity, as defined by the presence of receptor signaling in the absence of any ligand. Thus, ligands acting at a constitutively active receptor, can act as agonists, antagonists, and inverse agonists. In vitro studies have also revealed the complexity of ligand/receptor interactions including agonist-directed stimulus trafficking, a finding that has led to multi-active state models of receptor function. Studies with a variety of cell types have established that the serotonin 5-HT2A and 5-HT2C receptors also demonstrate constitutive activity and inverse agonism. However, until recently, there has been no evidence to suggest that these receptors also demonstrate constitutive activity and hence reveal inverse agonist properties of ligands in vivo. This paper describes our current knowledge of constitutive activity in vitro and then examines the evidence for constitutive activity in vivo. Both the seratonin 5-HT2A and 5-HT2C receptors are involved in a number of physiological and behavioral functions and are the targets for treatment of schizophrenia, anxiety, weight control, Parkinsonism, and other disorders. The existence of constitutive activity at these receptors in vivo, along with the possibility of inverse agonism, provides new avenues for drug development. (c) 2008 Elsevier Inc. All rights reserved.

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