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Diet controls Drosophila follicle stem cell proliferation via Hedgehog sequestration and release
Journal article   Open access   Peer reviewed

Diet controls Drosophila follicle stem cell proliferation via Hedgehog sequestration and release

Tiffiney R. Hartman, Todd I. Strochlic, Yingbiao Ji, Daniel Zinshteyn and Alana M. O'Reilly
The Journal of cell biology, v 201(5), pp 741-757
27 May 2013
PMID: 23690177
url
https://doi.org/10.1083/jcb.201212094View
Published, Version of Record (VoR) Open

Abstract

Cell Biology Life Sciences & Biomedicine Science & Technology
healthy diet improves adult stem cell function and delays diseases such as cancer, heart disease, and neurodegeneration. Defining molecular mechanisms by which nutrients dictate stem cell behavior is a key step toward understanding the role of diet in tissue homeostasis. In this paper, we elucidate the mechanism by which dietary cholesterol controls epithelial follicle stem cell (FSC) proliferation in the fly ovary. In nutrient-restricted flies, the transmembrane protein Boi sequesters Hedgehog (Hh) ligand at the surface of Hh-producing cells within the ovary, limiting FSC proliferation. Upon feeding, dietary cholesterol stimulates S6 kinase-mediated phosphorylation of the Boi cytoplasmic domain, triggering Hh release and FSC proliferation. This mechanism enables a rapid, tissue-specific response to nutritional changes, tailoring stem cell divisions and egg production to environmental conditions sufficient for progeny survival. If conserved in other systems, this mechanism will likely have important implications for studies on molecular control of stem cell function, in which the benefits of low calorie and low cholesterol diets are beginning to emerge.

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