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Genetic analysis of a mammalian wound-healing trait
Journal article   Open access

Genetic analysis of a mammalian wound-healing trait

Beth Ann McBrearty, Lise Desquenne Clark, Xiang-Ming Zhang, Elizabeth P Blankenhorn and Ellen Heber-Katz
Proceedings of the National Academy of Sciences - PNAS, v 95(20), pp 11792-11797
29 Sep 1998
PMID: 9751744
url
https://doi.org/10.1073/pnas.95.20.11792View
Published, Version of Record (VoR) Open

Abstract

Biological Sciences
Wound healing of mammalian tissue is an essential process in the maintenance of body integrity. The general mechanism of wound healing usually studied in adult mammals is repair, in contrast to the regeneration seen in more primitive vertebrates. We recently have discovered that MRL/MpJ mice, unlike all other strains of mice tested, undergo rapid and complete wound closure that resembles regeneration. Specifically, through-and-through surgical ear hole wounds close without scarring in <4 weeks with normal gross and microanatomic architecture, including chondrogenesis. We also demonstrated that this healing is a heritable trait in inbred mice. In this study, we present results pertaining to its genetic control in progeny segregating for this phenotype. To identify the genetic loci that control the wound closure process, a genome-wide scan was performed on (MRL/MpJ- Fas lpr × C57BL/6)F2 and backcross populations. In the primary screens of these populations, quantitative trait loci that control the extent of wound closure were detected on chromosomes 8, 12, and 15 and at two separate locations on chromosome 13. Evidence of further genetic control of healing was found on chromosome 7. All alleles that contribute to full wound closure are derived from the MRL/MpJ- Fas lpr parent except for the quantitative trait locus on chromosome 8, which is derived from C57BL/6.

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Collaboration types
Domestic collaboration
Web of Science research areas
Genetics & Heredity
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