Logo image
Genetic susceptibility to beryllium: a case-referent study of men and women of working age with sarcoidosis or other chronic lung disease
Journal article   Open access   Peer reviewed

Genetic susceptibility to beryllium: a case-referent study of men and women of working age with sarcoidosis or other chronic lung disease

Nicola Cherry, Jeremy Beach, Igor Burstyn, Jillian Parboosingh, Janine Schouchen, Ambikaipakan Senthilselvan, Larry Svenson, Jan Tamminga and Niko Yiannakoulias
Occupational and environmental medicine (London, England), v 72(1)
Jan 2015
PMID: 25305207
url
https://doi.org/10.1136/oemed-2014-102359View
Published, Version of Record (VoR) Open

Abstract

Genetic Predisposition to Disease Alberta Occupational Exposure - adverse effects Humans Middle Aged Genotype Male Sarcoidosis, Pulmonary - chemically induced HLA-DP beta-Chains - genetics Gene-Environment Interaction Asthma - chemically induced Asthma - genetics Case-Control Studies Young Adult Pulmonary Disease, Chronic Obstructive - chemically induced Adolescent Sarcoidosis, Pulmonary - genetics Adult Female Surveys and Questionnaires Beryllium - toxicity Chronic Disease Pulmonary Disease, Chronic Obstructive - genetics
The study was designed to investigate whether beryllium exposure was related to illness diagnosed as sarcoidosis. Chronic beryllium disease (CBD) and sarcoidosis are clinically and pathologically indistinguishable, with only the presence of beryllium-specific T-lymphocytes identifying CBD. Testing for such cells is not feasible in community studies of sarcoidosis but a second characteristic of CBD, its much greater incidence in those with a glutamic acid residue at position 69 of the HLA-DPB1 gene (Glu69), provides an alternative approach to answering this question. Cases of sarcoidosis aged 18-60 years diagnosed in Alberta, Canada, from 1999 to 2005 were approached through their specialist physician, together with age-matched and sex-matched referents with other chronic lung disease. Referents were grouped into chronic obstructive pulmonary disease (COPD), asthma and other lung disease. Participants completed a telephone questionnaire, including industry-specific questionnaires. DNA was extracted from mailed-in mouthwash samples and genotyped for Glu69. Duration of employment in types of work with independently documented beryllium exposure was calculated. DNA was extracted for 655 cases (270 Glu69 positive) and 1382 referents (561 positive). No increase in sarcoidosis was seen with either Glu69 or beryllium exposure (none, <10, ≥10 years) as main effects: longer duration in possible beryllium jobs was related to COPD. In Glu69 positive men with exposure ≥10 years, the trend towards increasing rate of COPD was reversed, and a significant interaction of duration of exposure and Glu69 was detected (OR=4.51 95% CI 1.17 to 17.48). The gene-environment interaction supports the hypothesis that some cases diagnosed as sarcoidosis result from occupational beryllium exposure.

Metrics

7 Record Views
13 citations in Scopus

Details

UN Sustainable Development Goals (SDGs)

This publication has contributed to the advancement of the following goals:

#3 Good Health and Well-Being

InCites Highlights

Data related to this publication, from InCites Benchmarking & Analytics tool:

Collaboration types
Domestic collaboration
International collaboration
Web of Science research areas
Public, Environmental & Occupational Health
Logo image