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Genome-wide DNA methylation analysis in cohesin mutant human cell lines
Journal article   Open access   Peer reviewed

Genome-wide DNA methylation analysis in cohesin mutant human cell lines

Jinglan Liu, Zhe Zhang, Masashige Bando, Takehiko Itoh, Matthew A. Deardorff, Jennifer R. Li, Dinah Clark, Maninder Kaur, Kondo Tatsuro, Antonie D. Kline, …
Nucleic acids research, v 38(17), pp 5657-5671
06 May 2010
PMID: 20448023
url
https://doi.org/10.1093/nar/gkq346View
Published, Version of Record (VoR) Open

Abstract

Gene Regulation, Chromatin and Epigenetics
The cohesin complex has recently been shown to be a key regulator of eukaryotic gene expression, although the mechanisms by which it exerts its effects are poorly understood. We have undertaken a genome-wide analysis of DNA methylation in cohesin-deficient cell lines from probands with Cornelia de Lange syndrome (CdLS). Heterozygous mutations in NIPBL, SMC1A and SMC3 genes account for ∼65% of individuals with CdLS. SMC1A and SMC3 are subunits of the cohesin complex that controls sister chromatid cohesion, whereas NIPBL facilitates cohesin loading and unloading. We have examined the methylation status of 27 578 CpG dinucleotides in 72 CdLS and control samples. We have documented the DNA methylation pattern in human lymphoblastoid cell lines (LCLs) as well as identified specific differential DNA methylation in CdLS. Subgroups of CdLS probands and controls can be classified using selected CpG loci. The X chromosome was also found to have a unique DNA methylation pattern in CdLS. Cohesin preferentially binds to hypo-methylated DNA in control LCLs, whereas the differential DNA methylation alters cohesin binding in CdLS. Our results suggest that in addition to DNA methylation multiple mechanisms may be involved in transcriptional regulation in human cells and in the resultant gene misexpression in CdLS.

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Biochemistry & Molecular Biology
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