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Glutamatergic systems in neuropathic pain and emerging non-opioid therapies
Journal article   Open access   Peer reviewed

Glutamatergic systems in neuropathic pain and emerging non-opioid therapies

Rhea Temmermand, James E. Barrett and Andreia C. K. Fontana
Pharmacological research, v 185, pp 106492-106492
01 Nov 2022
PMID: 36228868
url
https://doi.org/10.1016/j.phrs.2022.106492View
Accepted (AM)Open Access (Publisher-Specific) Open

Abstract

Life Sciences & Biomedicine Pharmacology & Pharmacy Science & Technology
Neuropathic pain, a disease of the somatosensory nervous system, afflicts many individuals and adequate management with current pharmacotherapies remains elusive. The glutamatergic system of neurons, receptors and transporters are intimately involved in pain but, to date, there have been few drugs developed that therapeutically modulate this system. Glutamate transporters, or excitatory amino acid transporters (EAATs), remove excess glutamate around pain transmitting neurons to decrease nociception suggesting that the modulation of glutamate transporters may represent a novel approach to the treatment of pain. This review highlights and summarizes (1) the physiology of the glutamatergic system in neuropathic pain, (2) the preclinical evidence for dysregulation of glutamate transport in animal pain models, and (3) emerging novel therapies that modulate glutamate transporters. Successful drug discovery requires continuous focus on basic and translational methods to fully elucidate the etiologies of this disease to enable the development of targeted therapies. Increasing the efficacy of astrocytic EAATs may serve as a new way to successfully treat those suffering from this devastating disease.

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29 citations in Scopus

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Collaboration types
Domestic collaboration
Web of Science research areas
Pharmacology & Pharmacy
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