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Identification of miR-185 as a regulator of de novo cholesterol biosynthesis and low density lipoprotein uptake
Journal article   Open access   Peer reviewed

Identification of miR-185 as a regulator of de novo cholesterol biosynthesis and low density lipoprotein uptake

Muhua Yang, Weidong Liu, Christina Pellicane, Christine Sahyoun, Biny K. Joseph, Christina Gallo-Ebert, Melissa Donigan, Devanshi Pandya, Caroline Giordano, Adam Bata, …
Journal of lipid research, v 55(2), pp 226-238
01 Feb 2014
PMID: 24296663
url
https://doi.org/10.1194/jlr.M041335View
Published, Version of Record (VoR) Open

Abstract

Biochemistry & Molecular Biology Life Sciences & Biomedicine Science & Technology
Dysregulation of cholesterol homeostasis is associated with various metabolic diseases, including atherosclerosis and type 2 diabetes. The sterol response element binding protein (SREBP)-2 transcription factor induces the expression of genes involved in de novo cholesterol biosynthesis and low density lipoprotein (LDL) uptake, thus it plays a crucial role in maintaining cholesterol homeostasis. Here, we found that overexpressing microRNA (miR)-185 in HepG2 cells repressed SREBP-2 expression and protein level. miR-185-directed inhibition caused decreased SREBP-2-dependent gene expression, LDL uptake, and HMG-CoA reductase activity. In addition, we found that miR-185 expression was tightly regulated by SREBP-1c, through its binding to a single sterol response element in the miR-185 promoter. Moreover, we found that miR-185 expression levels were elevated in mice fed a high-fat diet, and this increase correlated with an increase in total cholesterol level and a decrease in SREBP-2 expression and protein. Finally, we found that individuals with high cholesterol had a 5-fold increase in serum miR-185 expression compared with control individuals. Thus, miR-185 controls cholesterol homeostasis through regulating SREBP-2 expression and activity. In turn, SREBP-1c regulates miR-185 expression through a complex cholesterol-responsive feedback loop. Thus, a novel axis regulating cholesterol homeostasis exists that exploits miR-185-dependent regulation of SREBP-2 and requires SREBP-1c for function.

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Web of Science research areas
Biochemistry & Molecular Biology
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