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Impact of Tat Genetic Variation on HIV-1 Disease
Journal article   Open access   Peer reviewed

Impact of Tat Genetic Variation on HIV-1 Disease

Luna Li, Satinder Dahiya, Sandhya Kortagere, Benjamas Aiamkitsumrit, David Cunningham, Vanessa Pirrone, Michael R Nonnemacher and Brian Wigdahl
Advances in virology, v 2012, 123605
2012
PMID: 22899925
url
https://doi.org/10.1155/2012/123605View
Published, Version of Record (VoR) Open

Abstract

Review
The human immunodeficiency virus type 1 (HIV-1) promoter or long-terminal repeat (LTR) regulates viral gene expression by interacting with multiple viral and host factors. The viral transactivator protein Tat plays an important role in transcriptional activation of HIV-1 gene expression. Functional domains of Tat and its interaction with transactivation response element RNA and cellular transcription factors have been examined. Genetic variation within tat of different HIV-1 subtypes has been shown to affect the interaction of the viral transactivator with cellular and/or viral proteins, influencing the overall level of transcriptional activation as well as its action as a neurotoxic protein. Consequently, the genetic variability within tat may impact the molecular architecture of functional domains of the Tat protein that may impact HIV pathogenesis and disease. Tat as a therapeutic target for anti-HIV drugs has also been discussed.

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Web of Science research areas
Virology
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