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Increased Levels of Methylated Intermediates of Phosphatidylcholine Lead to Enhanced Phospholipase D Activity
Journal article   Peer reviewed

Increased Levels of Methylated Intermediates of Phosphatidylcholine Lead to Enhanced Phospholipase D Activity

Thomas Jacobs, Brent Passarello and Joel Horwitz
Neurochemical research, v 23(8), pp 1099-1105
Aug 1998
PMID: 9704600

Abstract

Biochemistry, general phosphatidyl-N-methylethanolamine Neurology Neurosciences Biomedicine phosphatidylcholine phospholipid methylation Phospholipase D PC12 cells
Previous work from this laboratory and others has shown that neurotransmitters can activate phospholipase D. Unlike the phospholipase C that specifically hydrolyzes inositol-containing phospholipids, phospholipase D in neuronal tissue specifically hydrolyzes phosphatidylcholine. One route for the synthesis of phosphatidylcholine, is via methylation of phosphatidylethanolamine. Using an in vitro assay, we have previously shown that methylated intermediates are also good substrates for phospholipase D (1). In this manuscript we demonstrate that these intermediates are also substrates in the intact PC 12 cells. Cells incubated with methyl and dimethylethanolamine incorporate more [3H]palmitic acid into the corresponding phospholipid, phosphatidyl-N-methylethanolamine and phosphatidyl-N,N-dimethylethanolamine. In these cells bradykinin causes a greater increase in [3H]phosphatidylethanol production. Elevated levels of [3H]phosphatidylcholine do not enhance bradykinin-stimulated [3H]phosphatidylethanol production, therefore, this effect is specific for the methylated intermediates. Finally, this effect is not due to some generalized enhancement of receptor coupling because incubation of the cells with methylethanolamine does not lead to an increase in bradykinin stimulated inositol phosphate production.

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Biochemistry & Molecular Biology
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