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Interactions between Swi1-Swi3, Mrc1 and S phase kinase, Hsk1 may regulate cellular responses to stalled replication forks in fission yeast
Journal article   Open access   Peer reviewed

Interactions between Swi1-Swi3, Mrc1 and S phase kinase, Hsk1 may regulate cellular responses to stalled replication forks in fission yeast

Michie Shimmoto, Seiji Matsumoto, Yukari Odagiri, Eishi Noguchi, Paul Russell and Hisao Masai
Genes to cells : devoted to molecular & cellular mechanisms, v 14(6), pp 669-682
01 Jun 2009
PMID: 19422421
url
https://doi.org/10.1111/j.1365-2443.2009.01300.xView
Published, Version of Record (VoR)Maybe Open Access (Publisher Bronze) Open

Abstract

Cell Biology Genetics & Heredity Life Sciences & Biomedicine Science & Technology
The Swi1-Swi3 replication fork protection complex and Mrc1 protein are required for stabilization of stalled replication forks in fission yeast. Hsk1 kinase also plays roles in checkpoint responses elicited by arrested replication forks. We show that both Swi1 and Swi3, the abundance of which are interdependent, are required for chromatin association of Mrc1. Co-immunoprecipitation experiments show the interactions of Swi1-Swi3, Mrc1 and Hsk1. Mrc1 interacts with Swi3 and Hsk1 proteins through its central segment (378-879) containing a SQ/TQ cluster, and this segment is sufficient for checkpoint reaction. The SQ/TQ cluster segment (536-673) is essential but not sufficient for the interactions and for resistance to replication inhibitor hydroxyurea. Mrc1 protein level is increased in hsk1-89 cells due to apparent stabilization, and we have identified a potential phosphodegron sequence. These results suggest that interactions of the Swi1-Swi3 complex and Hsk1 kinase with Mrc1 may play a role in cellular responses to stalled replication forks in fission yeast.

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Collaboration types
Domestic collaboration
International collaboration
Web of Science research areas
Cell Biology
Genetics & Heredity
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