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Mathematically engineered stromal cell-derived factor-1 alpha stem cell cytokine analog enhances mechanical properties of infarcted myocardium
Journal article   Open access   Peer reviewed

Mathematically engineered stromal cell-derived factor-1 alpha stem cell cytokine analog enhances mechanical properties of infarcted myocardium

John W. MacArthur, Alen Trubelja, Yasuhiro Shudo, Philip Hsiao, Alexander S. Fairman, Elaine Yang, William Hiesinger, Joseph J. Sarver, Pavan Atluri and Y. Joseph Woo
The Journal of thoracic and cardiovascular surgery, v 145(1), pp 278-284
01 Jan 2013
PMID: 23244259
url
https://doi.org/10.1016/j.jtcvs.2012.09.080View
Published, Version of Record (VoR)Open Access (Publisher-Specific) Open

Abstract

Cardiac & Cardiovascular Systems Cardiovascular System & Cardiology Life Sciences & Biomedicine Respiratory System Science & Technology Surgery
Objective: The biomechanical response to a myocardial infarction consists of ventricular remodeling that leads to dilatation, loss of contractile function, abnormal stress patterns, and ultimately heart failure. We hypothesized that intramyocardial injection of our previously designed pro-angiogenic chemokine, an engineered stromal cell-derived factor-1 alpha analog (ESA), improves mechanical properties of the heart after infarction. Methods: Male rats (n = 54) underwent either sham surgery (n = 17) with no coronary artery ligation or ligation of the left anterior descending artery (n = 37). The rats in the myocardial infarction group were then randomized to receive either saline (0.1 mL, n = 18) or ESA (6 mu g/kg, n = 19) injected into the myocardium at 4 predetermined spots around the border zone. Echocardiograms were performed preoperatively and before the terminal surgery. After 4 weeks, the hearts were explanted and longitudinally sectioned. Uniaxial tensile testing was completed using an Instron 5543 Microtester. Optical strain was evaluated using custom image acquisition software, Digi-Velpo, and analyzed in MATLAB. Results: Compared with the saline control group at 4 weeks, the ESA-injected hearts had a greater ejection fraction (71.8% +/- 9.0% vs 55.3% +/- 12.6%, P = .0004), smaller end-diastolic left ventricular internal dimension (0.686 +/- 0.110 cm vs 0.763 +/- 0.160 cm, P = .04), greater cardiac output (36 +/- 11.6 mL/min vs 26.9 +/- 7.3 mL/min, P = .05), and a lower tensile modulus (251 +/- 56 kPa vs 301 +/- 81 kPa, P = .04). The tensile modulus for the sham group was 195 +/- 56 kPa, indicating ESA injection results in a less stiff ventricle. Conclusions: Direct injection of ESA alters the biomechanical response to myocardial infarction, improving the mechanical properties in the postinfarct heart. (J Thorac Cardiovasc Surg 2013;145:278-84)

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Web of Science research areas
Cardiac & Cardiovascular Systems
Respiratory System
Surgery
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