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MicroRNAs, hepatitis C virus, and HCV/HIV-1 co-infection: new insights in pathogenesis and therapy
Journal article   Open access   Peer reviewed

MicroRNAs, hepatitis C virus, and HCV/HIV-1 co-infection: new insights in pathogenesis and therapy

Archana Gupta, Gokul Swaminathan, Julio Martin-Garcia and Sonia Navas-Martin
Viruses, v 4(11), pp 2485-2513
26 Oct 2012
PMID: 23202492
url
https://doi.org/10.3390/v4112485View
Published, Version of Record (VoR) Open

Abstract

Coinfection - therapy Hepatitis C - therapy Coinfection - genetics HIV-1 - physiology HIV Infections - genetics Humans HIV Infections - therapy Hepacivirus - physiology Hepatitis C - genetics MicroRNAs - metabolism MicroRNAs - genetics
MicroRNAs (miRNAs) can exert a profound effect on Hepatitis C virus (HCV) replication. The interaction of HCV with the highly liver-enriched miRNA, miR-122 represents one such unique example of viruses having evolved mechanism(s) to usurp the host miRNA machinery to support viral life cycle. Furthermore, HCV infection can also trigger changes in the cellular miRNA profile, which may ultimately contribute to the outcome of viral infection. Accumulating knowledge on HCV-host miRNA interactions has ultimately influenced the design of therapeutic interventions against chronic HCV infection. The importance of microRNA modulation in Human Immunodeficiency Virus (HIV-1) replication has been reported, albeit only in the context of HIV-1 mono-infection. The development of HCV infection is dramatically influenced during co-infection with HIV-1. Here, we review the current knowledge on miRNAs in HCV mono-infection. In addition, we discuss the potential role of some miRNAs, identified from the analyses of public data, in HCV/HIV-1 co-infection.

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Collaboration types
Domestic collaboration
Web of Science research areas
Virology
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