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Molecular Epidemiology of p53 Protein Mutations in Workers Exposed to Vinyl Chloride
Journal article   Peer reviewed

Molecular Epidemiology of p53 Protein Mutations in Workers Exposed to Vinyl Chloride

Steven J. Smith, Yongliang Li, Ronda Whitley, Marie-Jeanne Marion, Steven Partilo, Walter P. Carney and Paul W. Brandt-Rauf
American journal of epidemiology, v 147(3)
01 Feb 1998
PMID: 9482505

Abstract

epidemiology genes hemangiosarcoma liver neoplasms molecular occupational exposure p53 protein p53 vinyl chloride
The production of mutations in cellular tumor suppressor genes such as p53 is involved in the development of many human cancers. These mutations result in the expression of mutant forms of the encoded p53 protein which can potentially serve as a biomarker for this carcinogenic process. Workers exposed to vinyl chloride who are at risk for the development of the sentinel neoplasm angiosarcoma of the liver represent a model population for the study of such a mutant p53 biomarker, since vinyl chloride is known to cause specific p53 mutations in persons with angiosarcoma of the liver. To determine the relation between vinyl chloride exposure and this p53 biomarker, the authors examined serum samples collected between 1987 and 1992 from a cohort of 225 French vinyl chloride workers and 111 unexposed controls (matched according to age, sex, race, smoking, and alcohol drinking) for the presence of mutant p53 protein, using an enzyme-linked immunosor-bent assay. Stratification of the exposed workers by quartite of vinyl chloride exposure (in estimated ppm-years) yielded a statistically significant trend of increasing odds ratios for p53 biomarker seropositivity with increasing exposure. These results suggest that this serum biomarker for mutant p53 protein is related to vinyl chloride exposure and may be an early indicator of carcinogenic risk in exposed individuals. Am J Epidemiol 1998; 147: 302–8.

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Domestic collaboration
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Web of Science research areas
Public, Environmental & Occupational Health
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