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Molecular mechanisms of the preventable causes of cancer in the United States
Journal article   Open access   Peer reviewed

Molecular mechanisms of the preventable causes of cancer in the United States

Erica A. Golemis, Paul Scheet, Tim N. Beck, Eward M. Scolnick, David J. Hunter, Ernest Hawk and Nancy Hopkins
Genes & development, v 32(13-14), pp 868-902
01 Jul 2018
PMID: 29945886
url
http://genesdev.cshlp.org/content/32/13-14/868.full.pdfView
Published, Version of Record (VoR) Open
url
https://doi.org/10.1101/gad.314849.118View
Published, Version of Record (VoR) Open

Abstract

Cell Biology Developmental Biology Genetics & Heredity Life Sciences & Biomedicine Science & Technology
Annually, there are 1.6 million new cases of cancer and nearly 600,000 cancer deaths in the United States alone. The public health burden associated with these numbers has motivated enormous research efforts into understanding the root causes of cancer. These efforts have led to the recognition that between 40% and 45% of cancers are associated with preventable risk factors and, importantly, have identified specific molecular mechanisms by which these exposures modify human physiology to induce or promote cancer. The increasingly refined knowledge of these mechanisms, which we summarize here, emphasizes the need for greater efforts toward primary cancer prevention through mitigation of modifiable risk factors. It also suggests exploitable avenues for improved secondary prevention (which includes the development of therapeutics designed for cancer interception and enhanced techniques for noninvasive screening and early detection) based on detailed knowledge of early neoplastic pathobiology. Such efforts would complement the current emphasis on the development of therapeutic approaches to treat established cancers and are likely to result in far greater gains in reducing morbidity and mortality.

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Collaboration types
Domestic collaboration
International collaboration
Web of Science research areas
Cell Biology
Developmental Biology
Genetics & Heredity
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