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Monocyte-macrophage differentiation of acute myeloid leukemia cell lines by small molecules identified through interrogation of the Connectivity Map database
Journal article   Open access   Peer reviewed

Monocyte-macrophage differentiation of acute myeloid leukemia cell lines by small molecules identified through interrogation of the Connectivity Map database

Gloria Manzotti, Sandra Parenti, Giovanna Ferrari-Amorotti, Angela Rachele Soliera, Sara Cattelani, Monica Montanari, Daniel Cavalli, Adam Ertel, Alexis Grande and Bruno Calabretta
Cell cycle (Georgetown, Tex.), v 14(16), pp 2578-2589
18 Aug 2015
PMID: 26102293
url
https://doi.org/10.1080/15384101.2015.1033591View
Published, Version of Record (VoR)Maybe Open Access (Publisher Bronze) Open

Abstract

cell cycle differentiationm leukemia therapy transcription factor
The transcription factor C/EBPα is required for granulocytic differentiation of normal myeloid progenitors and is frequently inactivated in acute myeloid leukemia (AML) cells. Ectopic expression of C/EBPα in AML cells suppresses proliferation and induces differentiation suggesting that restoring C/EBPα expression/activity in AML cells could be therapeutically useful. Unfortunately, current approaches of gene or protein delivery in leukemic cells are unsatisfactory. However, "drug repurposing" is becoming a very attractive strategy to identify potential new uses for existing drugs. In this study, we assessed the biological effects of candidate C/EBPα-mimetics identified by interrogation of the Connectivity Map database. We found that amantadine, an antiviral and anti-Parkinson agent, induced a monocyte-macrophage-like differentiation of HL60, U937, Kasumi-1 myeloid leukemia cell lines, as indicated by morphology and differentiation antigen expression, when used in combination with suboptimal concentration of all trans retinoic acid (ATRA) or Vit D3. The effect of amantadine depends, in part, on increased activity of the vitamin D receptor (VDR), since it induced VDR expression and amantadine-dependent monocyte-macrophage differentiation of HL60 cells was blocked by expression of dominant-negative VDR. These results reveal a new function for amantadine and support the concept that screening of the Connectivity Map database can identify small molecules that mimic the effect of transcription factors required for myelo-monocytic differentiation.

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Web of Science research areas
Cell Biology
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