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Na +,K+-ATPase Activity and Subunit Isoform Protein Abundance: Effects of Antenatal Glucocorticoids in the Frontal Cerebral Cortex and Renal Cortex of Ovine Fetuses
Journal article   Peer reviewed

Na +,K+-ATPase Activity and Subunit Isoform Protein Abundance: Effects of Antenatal Glucocorticoids in the Frontal Cerebral Cortex and Renal Cortex of Ovine Fetuses

Najma S. Mehter, Grazyna B. Sadowska, Shadi N. Malaeb and Barbara S. Stonestreet
Reproductive sciences (Thousand Oaks, Calif.), v 16(3), 294
01 Mar 2009
PMID: 19001554

Abstract

dexamethasone Brain kidney development steroids sheep
We examined the effects of single and multiple maternal glucocorticoid courses on cerebral cortical (CC) and renal cortical (RC) Na+,K+-ATPase activity and protein isoform abundance in fetal sheep. Ewes received four dexamethasone or placebo injections in the single course (SC) groups, and the same treatment once a week for five-weeks in the multiple course (MC) groups. CC Na +,K+-ATPase a2-abundance was higher (P<0.05) and β2-abundance lower in the SC dexamethasone than placebo group, but Na+,K+-ATPase activity did not change. CC Na+,K+-ATPase activity, a1-, β1 -, and β2-abundance were lower in the MC dexamethasone than placebo group, but a2- and a3-abundance did not change. Both dexamethasone courses did not affect CC cell number. RC Na +,K+-ATPase activity, a1- and β1 -abundance were higher in the MC dexamethasone than placebo group, but did not change in the SC dexamethasone group. We conclude MC, but not a SC of dexamethasone, affect fetal cerebral and renal Na+,K+-ATPase, and MC result in differential effects on Na+,K+-ATPase in these organs.

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Collaboration types
Domestic collaboration
Web of Science research areas
Obstetrics & Gynecology
Reproductive Biology
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