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Nutrient sensor O-GlcNAc transferase controls cancer lipid metabolism via SREBP-1 regulation
Journal article   Open access   Peer reviewed

Nutrient sensor O-GlcNAc transferase controls cancer lipid metabolism via SREBP-1 regulation

Valerie L. Sodi, Zachary A. Bacigalupa, Christina M. Ferrer, Joyce V. Lee, Wiktoria A. Gocal, Dimpi Mukhopadhyay, Kathryn E. Wellen, Mircea Ivan and Mauricio J. Reginato
Oncogene, v 37(7), pp 924-934
23 Oct 2017
PMID: 29059153
url
https://europepmc.org/articles/pmc5814337View
Accepted (AM) Open

Abstract

ACLY cancer FAS lipid metabolism lipogenesis O-GlcNAc OGT SREBP-1
Elevated O-GlcNAcylation is associated with disease states such as diabetes and cancer. O-GlcNAc transferase (OGT) is elevated in multiple cancers and inhibition of this enzyme genetically or pharmacologically inhibits oncogenesis. Here we show that O-GlcNAcylation modulates lipid metabolism in cancer cells. OGT regulates expression of the master lipid regulator the transcription factor sterol regulatory element binding protein 1 (SREBP-1) and its transcriptional targets both in cancer and lipogenic tissue. OGT regulates SREBP-1 protein expression via AMP Activated protein kinase (AMPK). SREBP-1 is critical for OGT-mediated regulation of cell survival and of lipid synthesis, as overexpression of SREBP-1 rescues lipogenic defects associated with OGT suppression, and tumor growth in vitro and in vivo . These results unravel a previously unidentified link between O-GlcNAcylation, lipid metabolism and the regulation of SREBP-1 in cancer and suggests a crucial role for O-GlcNAc signaling in transducing nutritional state to regulate lipid metabolism.

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Collaboration types
Domestic collaboration
Web of Science research areas
Biochemistry & Molecular Biology
Cell Biology
Genetics & Heredity
Oncology
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