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O-GlcNAcylation in Cancer Biology: Linking Metabolism and Signaling
Journal article   Open access   Peer reviewed

O-GlcNAcylation in Cancer Biology: Linking Metabolism and Signaling

Christina M Ferrer, Valerie L Sodi and Mauricio J Reginato
Journal of molecular biology, v 428(16), pp 3282-3294
14 Aug 2016
PMID: 27343361
url
https://doi.org/10.1016/j.jmb.2016.05.028View
Published, Version of Record (VoR) Open

Abstract

Carcinogenesis - pathology Animals Humans Metabolic Networks and Pathways - physiology Signal Transduction - physiology Glycosylation Neoplasms - pathology Biosynthetic Pathways - physiology
The hexosamine biosynthetic pathway (HBP) is highly dependent on multiple metabolic nutrients including glucose, glutamine, and acetyl-CoA. Increased flux through HBP leads to elevated post-translational addition of β-D-N-acetylglucosamine sugars to nuclear and cytoplasmic proteins. Increased total O-GlcNAcylation is emerging as a general characteristic of cancer cells, and recent studies suggest that O-GlcNAcylation is a central communicator of nutritional status to control key signaling and metabolic pathways that regulate multiple cancer cell phenotypes. This review summarizes our current understanding of changes of O-GlcNAc cycling enzymes in cancer, the role of O-GlcNAcylation in tumorigenesis, and the current challenges in targeting this pathway therapeutically.

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Web of Science research areas
Biochemistry & Molecular Biology
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