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PRRC2A and BCL2L11 gene variants influence risk of non-Hodgkin lymphoma: results from the InterLymph consortium
Journal article   Open access   Peer reviewed

PRRC2A and BCL2L11 gene variants influence risk of non-Hodgkin lymphoma: results from the InterLymph consortium

Alexandra Nieters, Lucia Conde, Susan L. Slager, Angela Brooks-Wilson, Lindsay Morton, Danica R. Skibola, Anne J. Novak, Jacques Riby, Stephen M. Ansell, Eran Halperin, …
Blood, v 120(23), pp 4645-4648
29 Nov 2012
PMID: 23047821
url
https://ashpublications.org/blood/article-pdf/120/23/4645/987124/zh804912004645.pdfView
Published, Version of Record (VoR) Open
url
https://doi.org/10.1182/blood-2012-05-427989View
Published, Version of Record (VoR) Open

Abstract

Brief Report Lymphoid Neoplasia
Many common genetic variants have been associated with non-Hodgkin lymphoma (NHL), but individual study results are often conflicting. To confirm the role of putative risk alleles in B-cell NHL etiology, we performed a validation genotyping study of 67 candidate single nucleotide polymorphisms within InterLymph, a large international consortium of NHL case-control studies. A meta-analysis was performed on data from 5633 B-cell NHL cases and 7034 controls from 8 InterLymph studies. rs3789068 in the proapoptotic BCL2L11 gene was associated with an increased risk for B-cell NHL (odds ratio = 1.21, P random = 2.21 × 10 −11 ), with similar risk estimates for common B-cell subtypes. PRRC2A rs3132453 in the HLA complex class III region conferred a reduced risk of B-cell NHL (odds ratio = 0.68, P random = 1.07 × 10 −9 ) and was likewise evident for common B-cell subtypes. These results are consistent with the known biology of NHL and provide insights into shared pathogenic components, including apoptosis and immune regulation, for the major B-cell lymphoma subtypes.

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Collaboration types
Domestic collaboration
International collaboration
Web of Science research areas
Hematology
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