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Plasmodium chabaudi adami: interferon-γ but not IL-2 is essential for the expression of cell-mediated immunity against blood-stage parasites in mice
Journal article   Peer reviewed

Plasmodium chabaudi adami: interferon-γ but not IL-2 is essential for the expression of cell-mediated immunity against blood-stage parasites in mice

Joan M Batchelder, James M Burns, Francine K Cigel, Heather Lieberg, Dean D Manning, Barbara J Pepper, Deborah M Yañez, Henri van der Heyde and William P Weidanz
Experimental parasitology, v 105(2)
2003
PMID: 14969693

Abstract

Cell-mediated immunity (CMI) may be important in immunity against blood-stage malaria. Accordingly, we examined the role of type 1 cytokines in the resolution of Plasmodium chabaudi adami malaria in mice genetically modified to have type 1 cytokine gene defects. Parasitemia was prolonged in double knockout (IL-2 −/−, IFNγ −/−) mice compared to control mice. Despite deficiencies in γδ T cell and B cell subsets, these mice produced anti-malarial antibodies and eventually cured their infections, possibly by antibody-mediated immunity. However, because acute P. c. adami parasitemia may also be suppressed by CMI, the requirements for IL-2 and IFNγ were evaluated in mice lacking B cells and functional IL-2 or IFNγ genes. Acute malaria in J H −/−, IL-2 −/− mice was prolonged, but eventually cured. In contrast, J H −/−, IFNγ −/− mice developed unremitting parasitemia. These data strongly suggest that IFNγ, but not IL-2, plays an essential role in the expression of CMI against P. c. adami infections. This finding may prove useful in developing malarial vaccines aimed at inducing CMI. Index Descriptors and Abbreviations: Plasmodium chabaudi adami (P. c. adami); malaria; rodent; T H1/2 cytokines; infectious immunity-parasites; protozoan diseases; AMI, antibody-mediated immunity; CMI, cell-mediated immunity; KO, knockout; WT, wild-type; HRP, horse-radish peroxidase; B cells, B220 +/Ig + B cells; IL, interleukin

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Collaboration types
Domestic collaboration
Web of Science research areas
Parasitology
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