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Proliferation signal inhibitors and post-transplant malignancies in heart transplantation: practical clinical management questions
Journal article   Peer reviewed

Proliferation signal inhibitors and post-transplant malignancies in heart transplantation: practical clinical management questions

E. Epailly, J. Albanell, A. Andreassen, C. Bara, J. M. Campistol, J. F. Delgado, H. Eisen, A. E. Fiane, P. Mohacsi, S. Schubert, …
Clinical transplantation, v 25(5), pp E475-E486
01 Sep 2011
PMID: 21592231

Abstract

Life Sciences & Biomedicine Science & Technology Surgery Transplantation
Although malignancy is a major threat to long-term survival of heart transplant (HT) recipients, clear strategies to manage immunosuppression in these patients are lacking. Several lines of evidences support the hypothesis of an anticancer effect of proliferation signal inhibitors (PSIs: mammalian target of rapamycin [mTOR] inhibitors) in HT recipients. This property may arise from PSI's ability to replace immunosuppressive therapies that promote cancer progression, such as calcineurin inhibitors or azathioprine, and/or through their direct biological actions in preventing tumor development and progression. Given the lack of randomized studies specifically exploring these issues in the transplant setting, a collaborative group reviewed current literature and personal clinical experience to reach a consensus aimed to provide practical guidance for the clinical conduct in HT recipients with malignancy, or at high risk of malignancy, with a special focus on advice relevant to potential role of PSIs.

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Collaboration types
Domestic collaboration
International collaboration
Web of Science research areas
Surgery
Transplantation
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