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Replicase Genes of Murine Coronavirus Strains A59 and JHM Are Interchangeable: Differences in Pathogenesis Map to the 3′ One-Third of the Genome
Journal article   Open access   Peer reviewed

Replicase Genes of Murine Coronavirus Strains A59 and JHM Are Interchangeable: Differences in Pathogenesis Map to the 3′ One-Third of the Genome

Sonia Navas-Martin, Maarten Brom, Ming-Ming Chua, Richard Watson, Zhaozhu Qiu and Susan R Weiss
Journal of virology, v 81(2), pp 1022-1026
Jan 2007
PMID: 17079303
url
https://doi.org/10.1128/JVI.01944-06View
Published, Version of Record (VoR) Open

Abstract

Pathogenesis and Immunity
The important roles of the spike protein and other structural proteins in murine coronavirus (MHV) pathogenesis have been demonstrated; however, the role of the replicase gene remains unexplored. We assessed the influence of the replicase genes of the highly neurovirulent MHV-JHM strain and the hepatotropic and mildly neurovirulent A59 strain in acute infection of the mouse. Analysis of chimeric A59/JHM recombinant viruses indicates that the replicase genes are interchangeable and that it is the 3′ end of the genome, encoding the structural proteins, rather than the replicase gene, that determines the pathogenic properties of these chimeras.

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Collaboration types
Domestic collaboration
Web of Science research areas
Virology
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