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Repurposing ciclopirox as a pharmacological chaperone in a model of congenital erythropoietic porphyria
Journal article   Peer reviewed

Repurposing ciclopirox as a pharmacological chaperone in a model of congenital erythropoietic porphyria

Pedro Urquiza, Ana Lain, Arantza Sanz-Parra, Jorge Moreno, Ganeko Bernardo-Seisdedos, Pierre Dubus, Esperanza Gonzalez, Virginia Gutierrez-de-Juan, Sandra Garcia, Hasier Erana, …
Science translational medicine, v 10(459), 7467
19 Sep 2018
PMID: 30232228

Abstract

Cell Biology Life Sciences & Biomedicine Medicine, Research & Experimental Research & Experimental Medicine Science & Technology
Congenital erythropoietic porphyria is a rare autosomal recessive disease produced by deficient activity of uroporphyrinogen III synthase, the fourth enzyme in the heme biosynthetic pathway. The disease affects many organs, can be life-threatening, and currently lacks curative treatments. Inherited mutations most commonly reduce the enzyme's stability, altering its homeostasis and ultimately blunting intracellular heme production. This results in uroporphyrin by-product accumulation in the body, aggravating associated pathological symptoms such as skin photosensitivity and disfiguring phototoxic cutaneous lesions. We demonstrated that the synthetic marketed antifungal ciclopirox binds to the enzyme, stabilizing it. Ciclopirox targeted the enzyme at an allosteric site distant from the active center and did not affect the enzyme's catalytic role. The drug restored enzymatic activity in vitro and ex vivo and was able to alleviate most clinical symptoms of congenital erythropoietic porphyria in a genetic mouse model of the disease at subtoxic concentrations. Our findings establish a possible line of therapeutic intervention against congenital erythropoietic porphyria, which is potentially applicable to most of deleterious missense mutations causing this devastating disease.

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Collaboration types
Domestic collaboration
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Web of Science research areas
Cell Biology
Medicine, Research & Experimental
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