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Specific sequence configurations of HIV-1 LTR G/C box array result in altered recruitment of Sp isoforms and correlate with disease progression
Journal article   Peer reviewed

Specific sequence configurations of HIV-1 LTR G/C box array result in altered recruitment of Sp isoforms and correlate with disease progression

Michael R Nonnemacher, Bryan P Irish, Yujie Liu, David Mauger and Brian Wigdahl
Journal of neuroimmunology, v 157(1)
2004
PMID: 15579278

Abstract

HIV-1 LTR Sequence variation Markers
Basal and activated human immunodeficiency virus type 1 (HIV-1) long terminal repeat (LTR) activity, and in return, viral replication is partly dependent on interactions of the G/C box array with the Sp family of transcription factors. Analysis of LTR Sp binding site sequence variants revealed a C-to-T change at position 5 within Sp site III that increased in frequency and a 5T mutation within Sp site II, which decreased in frequency during the course of HIV disease. These results suggest LTR Sp binding site sequence variants may prove useful as viral molecular markers indicative of progressive HIV-1-induced disease.

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Collaboration types
Domestic collaboration
Web of Science research areas
Immunology
Neurosciences
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