Logo image
Stability and circularization of herpes simplex virus type 1 genomes in quiescently infected PC12 cultures
Journal article   Open access   Peer reviewed

Stability and circularization of herpes simplex virus type 1 genomes in quiescently infected PC12 cultures

Ying-Hsiu Su, Michael J Moxley, Alan K Ng, Judy Lin, Robert Jordan, Nigel W Fraser and Timothy M Block
Journal of general virology, v 83(Pt 12), pp 2943-2950
Dec 2002
PMID: 12466470
url
https://doi.org/10.1099/0022-1317-83-12-2943View
Published, Version of Record (VoR) Open

Abstract

Animals DNA, Viral - chemistry DNA, Viral - genetics DNA, Viral - metabolism Genome, Viral Herpesvirus 1, Human - genetics Herpesvirus 1, Human - pathogenicity Herpesvirus 1, Human - physiology Nerve Growth Factor - metabolism PC12 Cells - virology Rats Virus Activation Virus Latency Virus Replication
Herpes simplex virus type 1 (HSV-1) DNA has been shown to exist as a linear, double-stranded molecule in the virion and as a non-linear (endless), episomal, nucleosomal form in latently infected trigeminal ganglia. The kinetics of the formation and appearance of endless viral genomes and the stability of linear genomes in neuronal cells are not well understood. Nerve growth factor (NGF)-differentiated PC12 cells can sustain long-term, quiescent infections with HSV-1. In this report, the structure and stability of HSV-1 viral DNA in NGF-differentiated PC12 cells was studied as a function of time following infection using both wild-type and replication-defective virus. Unexpectedly, unencapsidated linear genomes were stable in the nucleus of NGF-differentiated PC12 cells for up to 2-3 weeks following infection, beyond the period at which there is no detectable viral gene expression. However, following infection with wild-type HSV, the majority of quiescent viral genomes were in an endless form after 3-4 weeks. These data suggest that the stability and fate of HSV-1 DNA in non-mitotic neuronal-like cells is different from that in productively infected cells and thus there is a significant cellular role in this process. The relevance to the virus life-cycle in neurones in vivo is discussed.

Metrics

6 Record Views
26 citations in Scopus

Details

UN Sustainable Development Goals (SDGs)

This publication has contributed to the advancement of the following goals:

#3 Good Health and Well-Being

InCites Highlights

Data related to this publication, from InCites Benchmarking & Analytics tool:

Collaboration types
Domestic collaboration
Web of Science research areas
Biotechnology & Applied Microbiology
Virology
Logo image