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Strategies, development, and pitfalls of therapeutic options for Alzheimer's disease
Journal article   Peer reviewed

Strategies, development, and pitfalls of therapeutic options for Alzheimer's disease

Christopher Haas
Journal of Alzheimer's disease, v 28(2)
2012
PMID: 21987594

Abstract

Alzheimer Disease - drug therapy Alzheimer Disease - metabolism Alzheimer Disease - pathology Alzheimer Disease - physiopathology Amyloid beta-Peptides - metabolism Antipsychotic Agents - therapeutic use Clinical Trials as Topic Humans Models, Biological Plaque, Amyloid - pathology tau Proteins - metabolism
Therapeutic options for Alzheimer's disease are currently limited to symptomatic treatment that only provides modest and temporary maintenance of cognitive and memory functions, without altering disease progression. Although a variety of therapeutics targeting amyloid production or plaque degradation as well as tau hyperphosphorylation and aggregation have been proposed, examined in pre-clinical models and introduced into clinical trials, many have failed to provide significant therapeutic benefit. Concerns over the adequacy of currently used pre-clinical models, in addition to questions pertaining to the timing of therapeutic administration, vis-à-vis synaptic and neuronal loss have been raised, and are further complicated by the genetic diversity of individual patients. This review will provide a brief overview of Alzheimer's disease pathophysiology and the currently approved therapeutics, while the main section will focus on therapeutics currently evaluated in pre-clinical models and clinical trials.

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