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Structure-based Design of Novel Small-Molecule Inhibitors of Plasmodium falciparum
Journal article   Open access   Peer reviewed

Structure-based Design of Novel Small-Molecule Inhibitors of Plasmodium falciparum

Sandhya Kortagere, William J Welsh, Joanne M Morrisey, Thomas Daly, Ijeoma Ejigiri, Photini Sinnis, Akhil B Vaidya and Lawrence W Bergman
Journal of chemical information and modeling, v 50(5), pp 840-849
24 May 2010
PMID: 20426475
url
https://doi.org/10.1021/ci100039kView
Published, Version of Record (VoR) Open

Abstract

hybrid structure based method myosin tail interacting protein gliding motility virtual screening plasmodium falciparum myosin A Antimalarials pyrazole urea
Malaria is endemic in most developing countries, with nearly 500 million cases estimated to occur each year. The need to design a new generation of antimalarial drugs that can combat the most drug-resistant forms of the malarial parasite is well recognized. In this study, we wanted to develop inhibitors of key proteins that form the invasion machinery of the malarial parasite. A critical feature of host-cell invasion by apicomplexan parasites is the interaction between the carboxy terminal tail of myosin A (MyoA) and the myosin tail interacting protein (MTIP). Using the co-crystal structure of the Plasmodium knowlesi MTIP and the MyoA tail peptide as input to the hybrid structure-based virtual screening approach, we identified a series of small molecules as having the potential to inhibit MTIP-MyoA interactions. Of the initial fifteen compounds tested, a pyrazole-urea compound inhibited P. falciparum growth with an EC 50 value of 145 nM. We screened an additional 51 compounds belonging to the same chemical class and identified eight compounds with EC 50 values less than 400 nM. Interestingly, the compounds appeared to act at several stages of the parasite’s life cycle to block growth and development. The pyrazole-urea compounds identified in this study could be effective antimalarial agents because they competitively inhibit a key protein-protein interaction between MTIP and MyoA responsible for the gliding motility and invasive features of the malarial parasite.

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Collaboration types
Domestic collaboration
Web of Science research areas
Chemistry, Medicinal
Chemistry, Multidisciplinary
Computer Science, Information Systems
Computer Science, Interdisciplinary Applications
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