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Structure of the MTIP–MyoA complex, a key component of the malaria parasite invasion motor
Journal article   Open access

Structure of the MTIP–MyoA complex, a key component of the malaria parasite invasion motor

Jürgen Bosch, Stewart Turley, Thomas M Daly, Stephen M Bogh, Michelle L Villasmil, Claudia Roach, Na Zhou, Joanne M Morrisey, Akhil B Vaidya, Lawrence W Bergman, …
Proceedings of the National Academy of Sciences - PNAS, v 103(13), pp 4852-4857
28 Mar 2006
PMID: 16547135
url
https://doi.org/10.1073/pnas.0510907103View
Published, Version of Record (VoR) Open

Abstract

Biological Sciences Plasmodium cell invasion machinery gliding motility myosin-tail-interacting protein
The causative agents of malaria have developed a sophisticated machinery for entering multiple cell types in the human and insect hosts. In this machinery, a critical interaction occurs between the unusual myosin motor MyoA and the MyoA-tail Interacting Protein (MTIP). Here we present one crystal structure that shows three different conformations of Plasmodium MTIP, one of these in complex with the MyoA-tail, which reveal major conformational changes in the C-terminal domain of MTIP upon binding the MyoA-tail helix, thereby creating several hydrophobic pockets in MTIP that are the recipients of key hydrophobic side chains of MyoA. Because we also show that the MyoA helix is able to block parasite growth, this provides avenues for designing antimalarials.

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Collaboration types
Domestic collaboration
Web of Science research areas
Biochemistry & Molecular Biology
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