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The CCK-A receptor antagonist, devazepide, blocks the anorectic action of CCK but not peripheral serotonin in rats
Journal article   Peer reviewed

The CCK-A receptor antagonist, devazepide, blocks the anorectic action of CCK but not peripheral serotonin in rats

K Eberle-Wang and K J Simansky
Pharmacology, biochemistry and behavior, v 43(3), pp 943-947
Nov 1992
PMID: 1448490

Abstract

Receptors, Cholecystokinin - antagonists & inhibitors Appetite Depressants - pharmacology Benzodiazepinones - pharmacology Cholecystokinin - pharmacology Rats Male Cholecystokinin - antagonists & inhibitors Devazepide Serotonin - pharmacology Serotonin Antagonists - pharmacology Rats, Sprague-Dawley Dose-Response Relationship, Drug Eating - drug effects Animals Diet
A role has been proposed for cholecystokinin (CCK)-A-type receptors in mediating the anorectic action produced by serotonergic stimulation in rats. We examined the effect of pretreatment with the CCK-A antagonist devazepide (DVZ) on anorexia produced by peripheral administration of serotonin [5-hydroxytryptamine (5-HT)] or CCK-8 in 3-h food-deprived rats consuming a 30-min test meal of sweetened mash. The anorectic effect of CCK-8 (4.0 nmol/kg, IP) was antagonized in a dose-dependent manner by DVZ (0.03, 0.10, and 0.30 mumol/kg, IP), with even the lowest dose producing a significant reversal. Under identical testing conditions, a supramaximal dose of DVZ (.75 mumol/kg) did not attenuate the reductions in food intake produced by either a moderate (4.0 mumol/kg) or a high dose (10.0 mumol/kg) of 5-HT. These data confirm established findings that the anorectic action of peripheral CCK depends upon CCK-A receptors. However, peripherally administered 5-HT reduces food intake independently of CCKergic function.

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Behavioral Sciences
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