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The compact conformation of the Plasmodium knowlesi myosin tail interacting protein MTIP in complex with the C-terminal helix of myosin A
Journal article   Open access   Peer reviewed

The compact conformation of the Plasmodium knowlesi myosin tail interacting protein MTIP in complex with the C-terminal helix of myosin A

Stewart Turley, Susmita Khamrui, Lawrence W Bergman and Wim G.J Hol
Molecular and biochemical parasitology, v 190(2)
Aug 2013
PMID: 23831369
url
https://doi.org/10.1016/j.molbiopara.2013.06.004View
Published, Version of Record (VoR) Open

Abstract

MTIP Malaria Glideosome X-ray structure Invasion
•The crystal structure of Plasmodium knowlesi MTIP in its compact conformation was solved.•This conformation differs dramatically from extended conformations we had determined before.•A neutral pH is important for MTIP to be able to adopt a compact conformation. The myosin motor of the malaria parasite's invasion machinery moves over actin fibers while it is making critical contacts with the myosin-tail interacting protein (MTIP). Previously, in a “compact” Plasmodium falciparum MTIP•MyoA complex, MTIP domains 2 (D2) and 3 (D3) make contacts with the MyoA helix, and the central helix is kinked, but in an “extended” Plasmodium knowlesi MTIP•MyoA complex only D3 interacts with the MyoA helix, and the central helix is fully extended. Here we report the crystal structure of the compact P. knowlesi MTIP•MyoA complex. It appears that, depending on the pH, P. knowlesi MTIP can adopt either the compact or the extended conformation to interact with MyoA. Only at pH values above ∼7.0, can key hydrogen bonds can be formed by the imidazole group of MyoA His810 with an aspartate carboxylate from the hinge of MTIP and a lysine amino group of MyoA simultaneously.

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Collaboration types
Domestic collaboration
Web of Science research areas
Biochemistry & Molecular Biology
Parasitology
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