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The mechanism of the negative chronotropic and dromotropic actions of adenosine 5'-triphosphate in the heart: an update
Journal article   Peer reviewed

The mechanism of the negative chronotropic and dromotropic actions of adenosine 5'-triphosphate in the heart: an update

Amir Pelleg and Bernard Belhassen
Journal of cardiovascular pharmacology, v 56(1), pp 106-109
Jul 2010
PMID: 20386456

Abstract

Adenosine - metabolism Adenosine Triphosphate - metabolism Adenosine Triphosphate - therapeutic use Electrophysiologic Techniques, Cardiac Heart Conduction System - drug effects Heart Conduction System - physiology Humans Syncope - drug therapy Syncope - physiopathology Tachycardia, Paroxysmal - drug therapy Tachycardia, Paroxysmal - physiopathology Tachycardia, Supraventricular - drug therapy Tachycardia, Supraventricular - physiopathology
Adenosine 5'-triphosphate (ATP) plays a critical role in intracellular metabolism and energetics. Extracellular ATP is rapidly degraded to adenosine by ectoenzymes. Both ATP and adenosine suppress cardiac pacemakers' automaticity and atrioventricular nodal conduction, albeit via the different mechanism of actions. This historical update summarizes the current knowledge regarding the negative chronotropic and dromotropic actions of ATP and discusses the clinical implications regarding the utility of ATP as a diagnostic and therapeutic agent in the management of neutrally mediated syncope and paroxysmal supra ventricular tachycardia.

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Collaboration types
Domestic collaboration
International collaboration
Web of Science research areas
Cardiac & Cardiovascular Systems
Pharmacology & Pharmacy
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