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The secretory phenotype of senescent astrocytes isolated from Wistar newborn rats changes with anti-inflammatory drugs, but does not have a short-term effect on neuronal mitochondrial potential
Journal article   Peer reviewed

The secretory phenotype of senescent astrocytes isolated from Wistar newborn rats changes with anti-inflammatory drugs, but does not have a short-term effect on neuronal mitochondrial potential

Luis Ángel Maciel-Barón, Sandra Lizbeth Morales-Rosales, Alejandro Silva-Palacios, Roxana Haydee Rodríguez-Barrera, Jorge Antonio García-Álvarez, Armando Luna-López, Viviana Isabel Pérez, Claudio Torres and Mina Königsberg
Biogerontology (Dordrecht), v 19(5), pp 415-433
Oct 2018
PMID: 30097900

Abstract

Animals Animals, Newborn Anti-Inflammatory Agents - pharmacology Astrocytes - drug effects Astrocytes - immunology Cellular Senescence - drug effects Cellular Senescence - immunology Central Nervous System - drug effects Central Nervous System - immunology Central Nervous System - metabolism Dehydroepiandrosterone - pharmacology Inflammation Interleukin-1alpha - immunology Isothiocyanates - pharmacology Membrane Potential, Mitochondrial - drug effects Models, Animal Neurons - drug effects Neurons - metabolism Oxidative Stress - physiology Rats Rats, Wistar
In the central nervous system (CNS), senescent astrocytes have been associated with neurodegeneration. Senescent cells secrete a complex mixture of pro-inflammatory factors, which are collectively called Senescence Associated Secretory Phenotype (SASP). The SASP components can vary depending on the cell type, senescence inducer and time. The SASP has been mainly studied in fibroblasts and epithelial cells, but little is known in the context of the CNS. Here, the SASP profile in senescent astrocytes isolated from Wistar newborn rats induced to senescence by oxidative stress or by proteasome inhibition was analyzed. Senescent astrocytes secreted predominantly chemokines and IL-1α, but no IL-6. The effect of the anti-inflammatory drugs, sulforaphane (SFN) and dehydroepiandrosterone (DHEA), on the SASP profile was evaluated. Our results showed that SFN and DHEA decreased IL-1α secretion while increasing IL-10, thus modifying the SASP to a less anti-inflammatory profile. Primary neurons were subjected to the conditioned media obtained from drug-treated senescent astrocytes, and their mitochondrial membrane potential was evaluated.

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Web of Science research areas
Geriatrics & Gerontology
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