Logo image
Thrombospondin-1 Is Elevated with both Intimal Hyperplasia and Hypercholesterolemia
Journal article   Peer reviewed

Thrombospondin-1 Is Elevated with both Intimal Hyperplasia and Hypercholesterolemia

Jeffrey J. Roth, Vivian Gahtan, Jeffrey L. Brown, Christine Gerhard, Vanlila K. Swami, Vicki L. Rothman, Thomas N. Tulenko and George P. Tuszynski
The Journal of surgical research, v 74(1), pp 11-16
Jan 1998
PMID: 9536966

Abstract

hypercholesterolemia intimal hyperplasia thrombospondin-1
Background.Thrombospondin-1 (TSP-1) is important in platelet adhesion and aggregation, inflammation, cell to cell interaction, angiogenesis, and smooth muscle cell (SMC) proliferation. TSP-1 expression increases rapidly with injury. Therefore, we hypothesize that TSP-1 may play a role in the development of intimal hyperplasia (IH). The purpose of this study is to examine the interaction between cholesterol and TSP-1 on SMC proliferation and to quantitatively assess TSP-1 expression in an established model of IH, with and without underlying cholesterol-induced atherosclerosis. Materials and methods. In vitro,rabbit aortic SMC culture studies were performed to see the effect of TSP-1 antibodies on PDGF and, separately, cholesterol-induced SMC proliferation.In vivo,23 rabbits were fed either a regular or a high-cholesterol diet. Hypercholesterolemia was confirmed by measurement of serum levels. Subsets underwent intraluminal aortic injury. Aortas were harvested 8–10 weeks later. Arterial wall TSP-1 was evaluated immunohistochemically and quantified by computer image analysis. Results. In vitro,TSP-1 antibodies were able to inhibit PDGF and cholesterol-induced SMC proliferation (P< 0.05).In vivo,TSP-1 was found predominantly in the extracellular matrix in the rabbit aorta. IH was uniformly seen status-post angioplasty. Hyperplasia was more prominent in samples from hypercholesterolemic animals. ANOVA and Student'sttest analyses demonstrated significantly more TSP-1 in the high-cholesterol/angioplasty group than in all other groups (P= 0.0006 vs regular diet/no angioplasty group). Conclusions.These data are consistent with the hypothesis that TSP-1 contributes to the development of IH. This study suggests that injured arteries in hypercholesterolemic atherosclerotic rabbits overexpress TSP-1.

Metrics

8 Record Views
89 citations in Scopus

Details

UN Sustainable Development Goals (SDGs)

This publication has contributed to the advancement of the following goals:

#3 Good Health and Well-Being

InCites Highlights

Data related to this publication, from InCites Benchmarking & Analytics tool:

Web of Science research areas
Surgery
Logo image