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Transient Left Ventricular Apical Ballooning After Cocaine Use: Is Catecholamine Cardiotoxicity the Pathologic Link?
Journal article   Open access   Peer reviewed

Transient Left Ventricular Apical Ballooning After Cocaine Use: Is Catecholamine Cardiotoxicity the Pathologic Link?

Sandeep Arora, Fadi Alfayoumi and Venkatraman Srinivasan
Mayo Clinic proceedings, v 81(6), pp 829-832
01 Jun 2006
DOI: https://doi.org/10.4065/81.6.829
PMID: 16770985
Featured in Collection :   UN Sustainable Development Goals @ Drexel
url
https://doi.org/10.4065/81.6.829View
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Abstract

ECG LVAB LVEF AMI LAD
We describe a patient who developed acute chest pain after using cocaine and had ST-segment elevation in the anterior leads on electrocardiography with mild elevation of cardiac enzymes. Cardiac catheterization showed normal coronary arteries with no coronary vasospasm. Left ventricular angiography revealed typical ballooning of the left ventricular apex during systole with an estimated left ventricular ejection fraction of 25%. The symptoms improved during the next few hours, and follow-up echocardiography 4 days later showed complete resolution of the left ventricular dysfunction. Transient left ventricular apical ballooning (LVAB) was diagnosed. To our knowledge, LVAB (also known as Takotsubo cardiomyopathy or “broken heart syndrome”) has not been reported previously in association with cocaine use. We discuss the possible pathophysiologic link between LVAB and cocaine-induced cardiotoxicity.

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72 citations in Web of Science
88 citations in Scopus

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UN Sustainable Development Goals (SDGs)

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#3 Good Health and Well-Being

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Collaboration types
Domestic collaboration
Web of Science research areas
Cardiac & Cardiovascular Systems
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