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Upregulation of adrenergic beta receptor subtypes in the senescent rat heart
Journal article   Peer reviewed

Upregulation of adrenergic beta receptor subtypes in the senescent rat heart

Nihal Tumer, Wayne T. Houck, Jay Roberts and James R Roberts
Mechanisms of ageing and development, v 49(3), pp 235-243
1989
PMID: 2554074

Abstract

Aging Beta-adrenergic receptor subtypes Heart Upregulation
Beta receptors in older hearts respond to procedures which cause upregulation (N. Turner et al., J. Gerontol., 1989, in press). To determine whether a particular beta receptor subtype is responsible for the development of upregulation as a function of age, we studied the radio of beta 1- to beta 2-adrenergic receptors in the membrane preparations from the ventricles of Fischer-344 hearts at 6 and 24 months of age. The animals were injected with 6-hydroxydopamine hydrobomide (6-OHDA) (2 × 50 mg/kg, i.v.) on days 1 and 8 and they were decapitated on day 15. The depletion of norepinephrine in the heart was about 86% in each age group. [ 125I]iodopindolol (IPIN) was used as the radioligand at the final concentration of 110 pM. Inhibition of specific IPIN binding was studied by adding ICI 89,406 (beta 1-selective antagnist) and ICI 118,551 (beta 2-selective antagonist) at 25 pM to 40μM. The relative proportions of the beta receptor subtypes were determined using a competition radioligand selective binding and computer modeling technique. The ventricles contained about 67% beta 1 and 33% beta 2-adrenergic receptors in hearts isolated from 6- and 24-month old rats; the ratio remained the same in sympathectomized animals. These data suggest that both subtypes of cardiac adrenergic receptors participate in the response to chemical denervation of 6-OHDA regardless of age.

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Web of Science research areas
Cell Biology
Geriatrics & Gerontology
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